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Characterized by lipid peroxidation and abnormal iron accumulation, ferroptosis has attracted significant attention due to its unique association with pathological conditions such as ischemia-reperfusion injury, neurodegeneration, and cancer [2,3,4]
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The sequences glutamine-X (any amino acid)-proline (QXP), QQ, and QXXQ are preferred targets for TGM2, whereas QP and QXXP are poor substrates (Fleckenstein et al., 2002
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