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Fasting-induced lipolysis is impaired in PEX5 AKO mice To elucidate the physiological roles of mammalian PEX5 upon nutritional deprivation, we decided to generate adipocyte-specific PEX5 knockout (PEX5 AKO) mice by crossing adiponectin-cre mice with PEX5-loxP mice in C57BL/6 background
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LM22A-4 has shown neuroprotective effects in Alzheimer cell culture models, and Dihexa restored learning and memory in rats with scopolamine-induced cognitive impairment
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