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Two of the six designs, pro1 and pro6, showed detectable binding to protegrin via changes in binder tryptophan fluorescence, with affinities of 7.2 and 1.2 µ M, respectively (Figure 3f, Supplementary Figure 22d)

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It has been observed that PD-1 blockade during primary infection increases viral loads, generates reactivations of other pathogens, increases tumor burden, and increases elevated production of immunosuppressive cytokines (301)